The distinction between idiopathic pulmonary arterial hypertension (iPAH) and pulmonary hypertension associated with lung disease remains increasingly challenging, particularly in patients with overlapping clinical features. In a timely study, Valentin and colleagues investigated whether cardiac magnetic resonance (CMR) native T1 mapping could improve phenotypic characterization in patients with classical iPAH, iPAH with a lung phenotype, and group 3 pulmonary hypertension (PH). While conventional CMR measures of right ventricular (RV) structure and function showed no significant differences between groups, native T1 values were significantly higher in classical iPAH, particularly at the interventricular septum and RV insertion points, suggesting more pronounced myocardial fibrosis and RV remodeling. Patients with lung phenotype iPAH exhibited T1 profiles closer to group 3 PH, supporting the concept of distinct underlying pathophysiology. Although the observed differences were modest and require validation across larger cohorts and 1.5 Tesla MRI platforms, these findings highlight the potential of myocardial tissue characterization as a complementary tool for integrated PH phenotyping. Beyond diagnostic classification, advanced CMR techniques may provide valuable biomarkers of RV adaptation, disease progression, and treatment response, contributing to a more personalized and mechanistically informed approach to pulmonary vascular disease.