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Archives of cardiovascular diseases
Volume 109, n° 8-9
pages 494-503 (août 2016)
Doi : 10.1016/j.acvd.2016.02.011
Received : 26 May 2015 ;  accepted : 4 February 2016
Consequences of obstructive sleep apnoea syndrome on left ventricular geometry and diastolic function
Conséquences du syndrome d’apnées du sommeil obstructif sur la géométrie et la fonction diastolique ventriculaire gauche
 

Figure 1




Figure 1 : 

Different types of left ventricular hypertrophy (LVH). A. Concentric LVH. B. Eccentric LVH. DIVGd: LV internal diameter, diastole; DIVGs: LV internal diameter, systole; PPVGd: LV posterior wall thickness, diastole; PPVGs: LV posterior wall thickness, systole; SIVd: interventricular septum thickness, diastole; SIVs: interventricular septum thickness, systole.


Figure 2




Figure 2 : 

Pathophysiological mechanisms involved in obstructive sleep apnoea syndrome-related left ventricular remodelling and diastolic dysfunction. RAAS: renin-angiotensin-aldosterone system.


Figure 3




Figure 3 : 

Diastolic dysfunction markers: A. Enlarged left atrium. B. Prolonged isovolumic relaxation time. C. Decreased E/A ratio. D. Restrictive mitral inflow pattern. E. Lower early diastolic mitral annular velocity (Ea in this figure). F. Increased E/e′ ratio. TRIV: isovolumic relaxation time; VM E Vit: early diastolic mitral peak flow velocity; VM T déc: E deceleration time; VM A Vit: late diastolic mitral peak flow velocity; Ea: early diastolic mitral annular velocity (e′).

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