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Archives of cardiovascular diseases
Volume 103, n° 8-9
pages 477-485 (août 2010)
Doi : 10.1016/j.acvd.2010.04.004
Received : 19 January 2010 ;  accepted : 2 April 2010
Gene therapy for heart failure
Thérapie génique pour l’insuffisance cardiaque
 

Figure 1




Figure 1 : 

Excitation-contraction signaling in cardiomyocytes with emphasis on targets for gene therapy. Interplay between calcium handling (purple) and β-adrenergic (blue) systems is illustrated. AC: adenylyl cyclase; ATP: adenosine triphosphate; β-AR: beta adrenergic receptor; I-1: (protein phosphatase) inhibitor-1; cAMP: cyclic adenosine monophosphate; Gs: stimulatory G protein; Gi: inhibitory G protein; NCX: sarcolemnal sodium/calcium exchanger; PKA: protein kinase A; PLN: phospholamban; PP: protein phosphatase; RyR: ryanodine receptor; SERCA: sarcoplasmic/endoplasmic reticulum calcium ATPase; SR: sarcoplasmic reticulum.

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