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Archives of cardiovascular diseases
Volume 106, n° 6-7
pages 366-372 (juin 2013)
Doi : 10.1016/j.acvd.2013.04.007
Received : 29 December 2012 ;  accepted : 10 April 2013
Dual phenotypic transmission in Brugada syndrome
Double transmission phénotypique dans le syndrome de Brugada
 

Figure 1




Figure 1 : 

First family pedigree. A. Baseline electrocardiogram. B. Electrocardiogram after ajmaline challenge. V1H, V2H, V1 and V2 lead at the second intercostal space. Proband: appearance of J waves in the inferior leads, amplitude of 1mm. BS: Brugada syndrome.


Figure 2




Figure 2 : 

Second family pedigree. A. Baseline electrocardiogram. B. Electrocardiogram after ajmaline challenge. V1H, V2H; V1 and V2 lead at the second intercostal space. Mother's electrocardiogram: at baseline, QTc 387ms; after ajmaline challenge, QTc 435ms. Father's electrocardiogram: at baseline, QTc 380ms; after ajmaline challenge, QTc 440ms. LQTS: long QT syndrome.


Figure 3




Figure 3 : 

Second family proband's electrocardiogram recorded 4days after cardiac arrest, without hypothermia or hyperthermia. QT interval 500ms; QTc interval 569ms. Nadolol 50mg/m2 was introduced 48hours before.


Figure 4




Figure 4 : 

Second family proband's electrocardiogram recorded 20days after cardiac arrest showing J wave, ST-segment elevation in V2 and V3 and long QTc interval (615ms). Patient under nadolol 50mg/m2.


Figure 5




Figure 5 : 

Electrogram recordings showing the onset of ventricular fibrillation and the appropriate shock delivered by the implanted cardiac defibrillator.

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