Elucidating the effects of disease-causing mutations on STAT3 function in autosomal-dominant hyper-IgE syndrome - 06/10/16
Doi : 10.1016/j.jaci.2016.04.020
Simon J. Pelham, MSc a, b, Helen C. Lenthall, MMSc b, Elissa K. Deenick, PhD a, b 
, Stuart G. Tangye, PhD a, b
, Stuart G. Tangye, PhD a, b a Immunology Research Program, Garvan Institute of Medical Research, Darlinghurst, Australia
b St Vincent's Clinical School, University of New South Wales Australia, Darlinghurst, Australia
| This work was supported by project and program grants awarded by the National Health and Medical Research Council (NHMRC) of Australia (grant nos. 1016953, 1066694, and 1027400 to E.K.D. and S.G.T.). S.J.P. is supported by an Australian Postgraduate Award from the University of New South Wales. S.G.T. is a recipient of a Principal Research Fellowship (1042925) from the NHMRC of Australia. |
|
| Disclosure of potential conflict of interest: S. J. Pelham has received a grant from the University of New South Wales (Australian Postgraduate Award) and the National Health Medical Research Council and has received travel support from Australian B Cell Dialogue 6. E. K. Deenick has received a grant from the National Health Medical Research Council and has received travel support from Baxalta. S. G. Tangye has received grants from the National Health Medical Research Council, has consultant arrangements with Eli Lilly, has served as an expert witness for a patent dispute, and has received travel support from Keystone Symposia. H. Lenthall declares no relevant conflicts of interest. |
© 2016
American Academy of Allergy, Asthma & Immunology. Publié par Elsevier Masson SAS. Tous droits réservés.
Vol 138 - N° 4
P. 1210 - octobre 2016 Retour au numéro
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