Genetic variants harbored in the forkhead box protein 3 locus increase hay fever risk - 07/08/11
Doi : 10.1016/j.jaci.2010.02.017
Kathrin Suttner, MSc a, Martin Depner, MSc b, Martin Wetzke, MD a, Norman Klopp, PhD c, Erika von Mutius, MD b, Thomas Illig, PhD c, Tim Sparwasser, MD d, Michael Kabesch, MD a 
a Center for Pediatrics, Clinic for Pediatric Pneumology and Neonatology, Hannover Medical School
b University Children’s Hospital, Ludwig Maximilian’s University, Munich
c Institute of Epidemiology, Helmholtz Center Munich, Neuherberg
d Institute of Infection Immunology, TWINCORE, Centre for Experimental and Clinical Infection Research, Hannover; a joint venture between the Helmholtz Centre for Infection Research (HZI) Braunschweig and the Hannover Medical School
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| Supported by the National Genome Research Network (NGFN) research grant NGFN 01GS 0810 and the German research foundation as part of the transregional collaborative research program TR22 “Allergic Immune Responses of the Lung,” grant A15/Z3. Genotyping was performed in the Genome Analysis Center of the Helmholtz Zentrum Munich. Funding sources had no influence in study design; in the collection, analysis, and interpretation of data; in the writing of the report; and in the decision to submit the paper for publication. |
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| Disclosure of potential conflict of interest: E. von Mutius is a consultant for GlaxoSmithKline, UCB, and ProtectImmun and has received research support from Airsonett AB. M. Kabesch has financial interests in Roxall, Glaxo Wellcome, Novartis, Sanofi Aventis, and Allergopharma and has received research support from the DFG, BMBF, and the European Union. The rest of the authors have declared that they have no conflict of interest. |
© 2010
American Academy of Allergy, Asthma & Immunology. Publié par Elsevier Masson SAS. Tous droits réservés.
Vol 125 - N° 6
P. 1395-1399 - juin 2010 Retour au numéro
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